Skip Navigation

This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Google Scholar
Right arrow Search for Related Content
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Continuing Education in Anaesthesia Critical Care and Pain | Volume 4 Number 3 | 2004
Continuing Education in Anaesthesia, Critical Care & Pain | Volume 4 Number 3 2004 © The Board of Management and Trustees of the British Journal of Anaesthesia 2004

Multiple Choice Questions

The first 10% of the full text of this article appears below.

50. The regulation of blood pressure:
  1. Principally involves the arterial baroreceptors on a minute-to-minute basis.
  2. Involves the renin–angiotensin–aldosterone but not the atrial natriuretic peptide system.
  3. Includes the regulation of renin solely by the concentration of sodium in the distal tubule.
  4. Is modulated by nitric oxide, a modulation that is impaired in hypertensive patients.
  5. Involves sodium balance in the long term.

51. The pathophysiology of hypertension:
  1. Necessarily includes an increase in both cardiac output and systemic vascular resistance.
  2. Most of the time includes a degree of vascular remodelling.
  3. Consistently includes a narrowing of the pulse pressure resulting from vascular remodelling.
  4. Is contributed to by increased autonomic activity and resetting of baroreflexes.
  5. Is not . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?