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Continuing Education in Anaesthesia, Critical Care & Pain 2006 6(1):42-44; doi:10.1093/bjaceaccp/mki070
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Continuing Education in Anaesthesia, Critical Care & Pain | Volume 6 Number 1 2006 © The Board of Management and Trustees of the British Journal of Anaesthesia [2006]. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Multiple Choice Questions

The first 10% of the full text of this article appears below.

1. Concerning the skeletal muscle sarcolemmal membrane potential:
  1. The cell is hyperpolarized by increased temperature.
  2. On depolarization, the Nernst potential for Na+ is reached.
  3. Cl– currents have no significant role under physiological conditions.
  4. A plateau potential is maintained by Ca2+ current through dihydropyridine channels.
  5. Repolarization is achieved principally by K+ current.

2. Excitation-contraction coupling in skeletal muscle:
  1. Involves a sarcolemmal Ca2+ current.
  2. Results in Ca2+ release through the SERCA pump.
  3. Involves interaction between dihydropyridine and ryanodine receptors.
  4. Is enhanced by high intracellular Mg2+ concentration.
  5. Is inhibited in malignant hyperthermia.

3. Concerning skeletal muscle proteins:
  1. Actin is the most abundant.
  2. Myosin is the largest.
  3. Myosin is the major constituent of thick filaments.
  4. Titin is a key structural element of thin filaments.
  5. Z proteins are formed from nebulin.

4. During skeletal muscle contraction:
  1. Unfolding of actin strands exposes the myosin binding sites.
  2. Ca2+ ions bind to troponin C.
  3. ATP is required . . . [Full Text of this Article]


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